To present the rationale and design for the PREVAIL trial (ALXN1720-MG-301; NCT05556096), a phase 3 study of ALXN1720 in adults with anti-acetylcholine receptor antibody-positive (AChR Ab+) generalized myasthenia gravis (gMG).
ALXN1720, a novel 28 kDa bispecific variable domain on a heavy chain antibody, binds complement component 5 (C5) inhibiting its cleavage of C5 into C5a and C5b and subsequent formation of the membrane attack complex. ALXN1720 also binds to albumin, which increases its half-life, facilitating weekly dosing. The low molecular weight of ALXN1720 enables its concentration in small volumes for subcutaneous (SC) delivery, allowing self-administration. Previous programs have demonstrated safety and efficacy of C5 inhibitors in gMG. A phase 3, randomized, double-blind, placebo-controlled study was designed to assess safety and efficacy of ALXN1720 in adults with AChR Ab+ gMG.
Approximately 200 patients will be randomized (1:1) to receive ALXN1720 or placebo. Patients will receive a weight-based loading dose of ALXN1720 or placebo, followed by weekly weight-based maintenance doses. PREVAIL consists of a 4-week screening period, a 26-week randomized controlled treatment phase and a 96-week open-label extension period. The efficacy of ALXN1720 versus placebo will be measured by changes from baseline at week 26 in Myasthenia Gravis-Activities of Daily Living (MG-ADL) total scores. Secondary outcome measures include changes from baseline at week 26 in Quantitative Myasthenia Gravis (QMG) and Myasthenia Gravis Composite total scores, and MG-ADL 3-point improvement and QMG 5-point improvement. The safety, pharmacokinetics, pharmacodynamics and immunogenicity of ALXN1720 will be assessed. Key eligibility criteria include documented diagnosis of gMG ≥3 months ago; positive serological test for AChR autoantibodies; Myasthenia Gravis Foundation of America disease classification II to IV; and MG-ADL total score ≥5.
PREVAIL is currently active and recruiting patients in multiple countries.
PREVAIL will assess safety and efficacy of SC ALXN1720 in patients with AChR Ab+ gMG.