Determinants of Refractory Course and Outcomes in Adult Status Epilepticus at a Regional Center
Curtis Amankwah1, Anastasiia Strelkova1, Mitchell Dubaniewicz1, Amr Ewida2
1Neurology, West Virginia University, 2West Virginia University School of Medicine
Objective:

To identify clinical and treatment factors associated with refractory SE (RSE), in-hospital mortality, and resource use in an adult SE cohort.

Background:

Status epilepticus (SE) carries substantial morbidity and mortality in both classic population studies and contemporary regional cohorts.

Design/Methods:

Retrospective cohort of consecutive adults with chart-confirmed SE over one year. Variables included demographics, prior epilepsy, onset location, etiology, semiology, EEG findings, prehospital/ED benzodiazepines (BZDs), first antiseizure medication (ASM), GCS, LOS/ICU days, and discharge outcomes. Analyses: multivariable logistic regression for RSE; penalized logistic regression for mortality (to address separation with GCS/etiology); Gamma GLM for LOS.

Results:

We included 81 patients (52% female; median age 62 years). RSE occurred in 36 (44.4%), and in-hospital mortality was 22.2% (18). In adjusted analyses:

·       A history of epilepsy was associated with reduced odds of RSE (aOR 0.15, p=0.022).

·       Convulsive semiology (aOR 4.32, p=0.040) and EEG epileptiform abnormalities (aOR 3.91, p=0.046) were associated with increased odds of RSE.

·       Compared to lorazepam, midazolam showed a trend toward reduced RSE risk (aOR 0.26, p=0.064).

·       In the penalized mortality model, deaths were confined to acute etiologies (notably anoxic injury and cerebrovascular events) and low GCS (≤8).

·       Myoclonic semiology was strongly associated with mortality (OR 25.0, 95% CI 5.7–110.5, p<0.001). In-hospital onset and acute etiologies were associated with mortality risk, while male sex, prior epilepsy, and convulsive semiology were linked to lower risk.

·       Longer stays were associated with EEG epileptiform abnormalities (~65% increase, p=0.044) and older age (~1.7% per year, p=0.024).


 

Conclusions:

In this cohort, independent determinants of RSE included prior epilepsy (protective), convulsive semiology, and EEG epileptiform abnormalities. Mortality was driven by acute etiologies (anoxia and cerebrovascular events), low GCS, and in-hospital onset. EEG abnormalities and age increased resource use. These findings complement historical and contemporary series and highlight targets for prognostication and protocol optimization.

10.1212/WNL.0000000000217400
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