Paralysis Associated With Coccidioidal Meningitis: A Review of 34 Cases
Safa Mousavi1, Divanshu Sharma1, Bianca Torres1, Jigar Patel2, Michelle Fang3, Shikha Mishra2, Rasha Kuran2, Royce Johnson2, Carlos DAssumpcao2
1Valley Fever Institute, Kern Medical, 2Department of Medicine, Division of Infectious Disease, 3Department of Pharmacy, Kern Medical
Objective:

To describe the clinical presentation, neuroradiology findings, treatment, and outcomes in paralyzed patients with Coccidioidal meningitis

Background:

Coccidioidal meningitis (CM) is a life-threatening condition and a diagnostic challenge. It is associated with infarction, aneurysms, hydrocephalus, intracranial hemorrhage, transient ischemic attacks, syrinx, and arachnoiditis. Untreated, it is fatal within 2 years.

Design/Methods:

We conducted a retrospective case review (2011–2023) identified by ICD-9/10 codes and positive Coccidioides IgG/IgM in CSF. Confirmed CM, paralysis, and available neuroimaging were included. Patients under 18 years or those with insufficient data were excluded.

Results:

Thirty-four patients were identified; mean age was 45.5 years (range: 21 to 64 years old). Twenty-four patients were male, and most were White Hispanic. Twenty-nine patients developed paralysis on therapy for CM, while 5 presented with paralysis as the initial manifestation of CM.

Headache was the most common initial symptom. Paralysis developed after a median of 18 months (range: 6 days–19 years) from initial CM diagnosis. Twenty-one patients were treated initially with fluconazole, 16 of whom were transitioned to broad-spectrum triazoles at the onset of paralysis: 1 to itraconazole, 7 to voriconazole, 2 to posaconazole, and 6 to isavuconazole. At paralysis onset, along with azole therapy, adjunctive therapy included intravenous liposomal amphotericin B, intrathecal amphotericin B, in combination with dexamethasone. Twelve patients had ventriculoperitoneal (VP) shunts placed: 7 at the time of CM diagnosis and 5 at the onset of paralysis.

Neuroradiologic findings at paralysis onset included basilar arachnoiditis (16/34), hydrocephalus (11/34), vasculitic infarctions at various locations (19/34), spinal arachnoiditis (16/34), syrinx (3/34), and subarachnoid hemorrhage (3/34). One patient had a right middle cerebral artery aneurysm.

Twenty-eight patients survived with the significant morbidity, and 6 patients died.
Conclusions:

Paralysis in patients with CM is associated with significant morbidity and mortality. Antifungal therapy with adjunctive glucocorticoids reduces mortality rate.

10.1212/WNL.0000000000215698
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