A woman in her 20s presented for evaluation of headaches with nausea/photophobia and episodic confusion/disorientation. Initially diagnosed with rhinosinusitis, her symptoms continued to worsen despite treatment.
MRI brain with/without contrast revealed acute and subacute lacunar infarcts in the right frontal lobe, corpus callosum, and left parietal lobe. CT angiogram and echocardiogram with bubble were normal. Hypercoagulable workup and body CT screening for malignancy were negative. Long-term EEG showed no epileptiform activity. Cerebrospinal fluid (CSF) analysis demonstrated elevated protein (182), normal glucose/cellularity, and negative cultures. Headaches and episodic encephalopathy continued to worsen. Repeat MRI brain demonstrated new callosal and infratentorial lacunar infarcts. CSF protein increased on repeat analysis, but autoantibody panel resulted negative.
Lesion location (predominantly small vessel with callosal involvement), elevated CSF protein, and lack of typical vascular risk factors prompted concern for SS. Initial in-hospital audiology and ophthalmology evaluations were unrevealing; however, retinal fluorescein angiography (RFA) revealed binocular microangiopathy and BRAO. She was given methylprednisolone with symptomatic improvement and continues on maintenance IVIG therapy.